Prediction of breast cancer risk by genetic risk factors, overall and by hormone receptor status

Knowing a woman’s risk to develop breast cancer at a given age can be an important tool for optimized targeting of preventive measures such as screening or chemo prevention. In a large population study, we examined if 32 genetic markers that were previously identified through systematic genetic searches can improve the prediction of breast cancer […]

Read More…

Deletion of the 3q26 region including the EVI1 and MDS1 genes in a neonate with congenital thrombocytopenia and subsequent aplastic anaemia

We describe for the first time a patient who has only one copy of the EVI1- and MDS1-gene, because of an inborn chromosomal defect. These two genes are known to be important for the stem cell functioning of the bone marrow, and thereby essential for the ongoing formation of blood cells. The decrease in the […]

Read More…

Complex I deficiency: clinical features, biochemistry and molecular genetics

The challenges of genetically diagnosing complex I deficiency, the most common cause of childhood-onset mitochondrial disease, are not only due to the very broad spectrum of clinical presentations, but also because of the large number of potential disease-causing genes. This review discusses the structure and function of complex I in light of recent X-ray crystallographic […]

Read More…

Recessive germline SDHA and SDHB mutations causing leukodystrophy and isolated mitochondrial complex II deficiency

Mitochondrial diseases affect both children and adults and are clinically and genetically diverse, on account of the dual genetic control of mitochondrial function. Isolated complex II deficiency is a rare form of mitochondrial disease, due to autosomal recessive mutations in either structural components of this enzyme (the SDHA, SDHB, SDHC and SDHD genes) or two […]

Read More…

Exome sequencing identifies a COL14A1 mutation in a large Chinese pedigree with punctate palmoplantar keratoderma

Punctate palmoplantar keratoderma (PPPK) is a rare autosomal dominant skin disorder characterized by numerous hyperkeratotic papules irregularly distributed on the palms and soles. To date, no causal gene for this disease has been identified. The authors identified a novel heterozygous mutation (c.4505C->T [p.Pro1502Leu]) in COL14A1 gene using exome sequencing, which is a missense substitution at […]

Read More…

Dominantly inherited diabetes mellitus caused by GATA6 haploinsufficiency: variable intrafamilial presentation

So far, abnormality of the GATA6 gene has been linked to only two clinical conditions: congenital heart malformation and recently described pancreatic agenesis. Previously reported cases of GATA6 pancreatic disease were sporadic and born with a total or near-total absence of the pancreas, most of them accompanied by severe neonatal diabetes mellitus and heart malformation. […]

Read More…

Quantitative trait locus analysis for next-generation sequencing with the functional linear models

It is now well documented that next-generation sequencing (NGS) can generate several millions or even dozens of millions of genetic variation data. As a consequence, these genetic variation data are so densely distributed across the genome that the genetic variation can be modeled as a function of genomic location. But, standard multivariate statistical analysis often […]

Read More…

A systematic review of associated structural and chromosomal defects in oral clefts: when is prenatal genetic analysis indicated?

Oral clefts are being diagnosed prenatally more frequently. When informing future parents on outcome and prognosis, the category of cleft as well as the presence of other structural congenital anomalies is crucial. Especially the identification of an underlying chromosomal defect will influence prenatal counselling and management of the pregnancy significantly. To provide a basis for […]

Read More…

Clinical significance of copy number variations in the 11p15.5 imprinting control regions: new cases and review of the literature

The chromosomal region 11p15 harbours several imprinted genes, i.e. genes which are expressed in a parent-of-origin manner. Opposite molecular disturbances in 11p15 are associated with two distinct disorders associated with disturbed growth, Silver-Russell and Beckwith-Wiedemann syndrome. Due to the complexity of the 11p15 region and the interactions between the different genes, the interpretation of copy […]

Read More…