A syndrome of congenital hyperinsulinism and rhabdomyolysis is caused by KCNJ11 mutation

KCNJ11 encodes an important component of the potassium channel that plays a critical role in regulating the response of beta cells in the pancrease to changes in blood glucose level.  Mutations in this gene can cause inappropriately reduced or elevated insulin secretion.  In a large and extended consanguineous Saudi family, we show for the first […]

Read More…

Recurrent X chromosome-linked deletions: discovery of new genetic factors in male infertility

The etiology of altered spermatogenesis is  unknown in about 40% of cases and a large proportion of it is likely related to still unknown genetic factors. The identification of  the “missing” genetic causes is of importance both for genetic counseling and for the development  of  future etiologic therapies. Our study, by screening over 1200 subjects […]

Read More…

Prenylation defects in inherited retinal diseases

To fulfil their specialised functions, retinal proteins are located at specific positions in photoreceptor cells. These cells consist of many different compartments. Proteins are transported between these compartments through their binding to other proteins or to membranous vesicles. To interact with other proteins or vesicles, proteins contain a hydrophobic lipid molecule, a prenyl group. Many […]

Read More…

Methyl-CpG-binding protein 2 (MECP2) mutation type is associated with disease severity in Rett syndrome

Using the largest patient cohort studied (1052 patients) with Rett syndrome (RTT), we investigated whether individual mutations give rise to the observed phenotypic variability.  We identified groups of point mutations, deletions and insertions that were particularly severe in both typical and atypical RTT, as well as groups of mutations were less severe.  Our data suggest […]

Read More…

The novel mitochondrial 16S rRNA 2336T>C mutation is associated with hypertrophic cardiomyopathy

Hypertrophic cardiomyopathy (HCM) is a significant cause of sudden unexpected cardiac death in any age group. Here we reported a Chinese family with maternally transmitted HCM and atrioventricular block (AVB). DNA sequencing analysis identified a novel homoplasmic 2336T>C mutation, which disturbs the 2336U-A2438 base pair in the stem-loop structure of mitochondrial 16S rRNA domain III. […]

Read More…

The HNF4A R76W mutation causes atypical dominant Fanconi syndrome in addition to a beta cell phenotype

The authors have discovered that a specific mutation in the HNF4A gene causes Fanconi syndrome, where salt and protein loss by the kidneys leads to soft bones and short stature. People with this condition usually have to take tablets to replace the salts leaked by the kidneys, and the genes responsible haven’t been found until […]

Read More…

Paralogue annotation identifies novel pathogenic variants in patients with Brugada syndrome and Catecholaminergic Polymorphic Ventricular Tachycardia

As genetic testing of inherited diseases becomes ever more widely used, distinguishing between true disease-causing mutations and innocuous rare genetic variants is a critical issue in clinical genetics. Our new analysis method transfers known clinical mutations between evolutionarily related proteins, or paralogues, allowing us to identify mutations that affect parts of the protein intolerant to […]

Read More…

Autism traits in the RASopathies

This ‘pathway’ approach investigates the Ras/MAPK signaling role in autism traits.  A large sample representing genetic disorders (RASopathies) caused by mutations activating this pathway was compared with sibling controls and autism spectrum disorder (ASD) subjects.  Each RASopathy (Neurofibromatosis type 1, Noonan syndrome, Costello syndrome, and cardio-facio-cutaneous syndrome) is associated with autism traits, with distinct distributions.  […]

Read More…

Direct-to-consumer pharmacogenomic testing is associated with increased physician utilisation

The impact of direct-to-consumer (DTC) pharmacogenomic testing on consumer behavior has not been previously studied.  A large sample of adults purchased a DTC pharmacogenomic test and completed baseline and follow-up health behavior assessments.  The pharmacogenomic test included testing for drug effectiveness or risk of side effects for 12 medications.  At follow-up the 481 individuals who […]

Read More…